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HOME > Endocrinol Metab > Volume 25(4); 2010 > Article
Case Report A Case of Thyroid Amyloidosis Misrecognized as Subacute Thyroiditis.
Young Sil Lee, Seong Su Moon, Soon Kim, Jong Im Lee
Endocrinology and Metabolism 2010;25(4):360-364
DOI: https://doi.org/10.3803/EnM.2010.25.4.360
Published online: December 1, 2010
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1Department of Internal Medicine, Dongguk University College of Medicine, Gyeongju, Korea. ysbae28@medimail.co.kr
2Department of Diagnostic Radiology, Dongguk University College of Medicine, Gyeongju, Korea.
3Department of Pathology, Dongguk University College of Medicine, Gyeongju, Korea.
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Abstract

Amyloid deposition in the thyroid is found in about 30-80% of the patients with primary or secondary amyloidosis. In a few patients with amyloidosis, the thyroid is enlarged and so called amyloid goiter occurs. The thyroid function usually remains normal, but occasionally hypothyroidism or thyrotoxicosis occurs in patients with thyroid amyloidosis. We have experienced one case of thyroid amyloidosis due to bronchiectasis and the patient developed painful goiter and transient thyrotoxicosis resembling subacute thyroiditis. Biopsies from the gastric antrum, duodenum, colon, kidney and thyroid were positive for deposition of amyloid. When patients have signs and symptoms of subacute thyroiditis, but they develop an unusual course, then the diagnosis of thyroid amyloidosis should be considered.

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