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Endocrinol Metab : Endocrinology and Metabolism


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Mi Jin Kim  (Kim MJ) 4 Articles
A Case of Drug Induced Nephrogenic Diabetes Insipidus and Hyperprolactinemia in Schizophrenia Simultaneously.
Ho Yoel Ryu, Mi Young Lee, Yeon Lee, Jang Hyun Koh, Mi Jin Kim, Young Goo Shin, Choon Hee Chung
J Korean Endocr Soc. 2005;20(4):407-412.   Published online August 1, 2005
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  • 42 Download
AbstractAbstract PDF
In schizophrenia, when treatment using antipsychotics fails, lithium, which is known as an antimanic drug, can also be administered. It is reported that 12~20% of patients taking lithium develop nephrogenic diabetes lactotrophs. Hyperprolactinemia is induced by typical antipsychotics, as they block the dopamine-2 receptors of latotrophs in the pituitary gland. Therefore, atypical antipsychotics for decreasing the side effect, such as hyperprolactinemia, can be used. However, hyperprolactinemia can be induced by risperidone, one of the atypical antipsychotics. Here, a case of drug induced nephrogenic diabetes insipidus and simultaneous hyperprolactinemia, which occurred in a patient with schizophrenia, is reported.
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Two Cases of Apathetic Hyperthyroidism associated with Peripheral Eosinophilia.
Mi Jin Kim, Kwang Ha Yoo, Byung Kook Kim, Won Chul Chang, Jai Ho Yeon, Sang Youl Shin
J Korean Endocr Soc. 2005;20(1):78-83.   Published online February 1, 2005
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  • 40 Download
  • 1 Crossref
AbstractAbstract PDF
Peripheral eosinophilia is perpetually associated with many disease conditions like asthma, parasitic disease, IL-2 therapy, hypereosinophilic syndrome and eosinophilia/myalgia syndrome. Furthermore, peripheral eosinophilia may also be linked with Graves' disease, which is induced by thyroid-stimulating anti-TSH receptor antibodies and is related to type 2 helper T cell(Th2) predominant condition that is dependent on humoral immunity. In some of the cases of Graves' disease, thyrotoxicosis may induce peripheral eosinophilia associated with relative cortisol deficiency. In our present study, we present a case of two patients who were suffering from Graves' disease without any thyrotoxic symptoms and exhibited increased levels of peripheral eosinophil count. But, the count was observed to decreas in euthyroid state.


Citations to this article as recorded by  
  • Case Report: A Rare Case of Eosinophilic Ureteritis in a Woman with Reccuring Bilateral Ureteric Strictures causing Hydroureteronephrosis
    Sahat Basana Romanti Ezer Matondang, Adistra Imam, Guntur Darmawan, Armand Achmadsyah, Ibrahim Abubakar Hilmy, Andrian Harsanto, Rochani Sumardi
    F1000Research.2023; 12: 248.     CrossRef
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A Case of Pseudopseudohypoparathyroidism with Partial Empty Sella.
Jang Hyun Koh, Yeon Lee, Joung Wook Choi, Tae Won Hong, Mi Jin Kim, Young Goo Shin, Choon Hee Chung
J Korean Endocr Soc. 2004;19(4):433-438.   Published online August 1, 2004
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  • 18 Download
AbstractAbstract PDF
Pseudohypoparathyroidism (PHP) is a rare endocrine syndrome, which is characterized by Albright's hereditary osteodystrophy (AHO). Pseudopseudohypoparathyroidism (PPHP) presents with AHO features, but does not show clinical symptoms or the same laboratory finding as hypoparathyroidism. A 54 aged woman visited our department with lower abdominal pain. She had a history of a total hysterectomy due to persistent uterine bleeding 30 years previously. She showed abnormal physical findings, including a round face, short stature and brachydactyly. Her hormonal data were within normal limits, as follows: PTH 40.72 pg/mL, calcium 8.6 mg/dL, phosphorus 4.0 mg/dL and 24 hour urine cAMP 3.2 nmol/mg. On a pituitary challenge test, the basal levels and response rates of FSH and LH were low, and a sella CT scan showed partially empty sella. Herein is reported a case of PPHP, with partial empty sella, with a review of the literature
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A Case of X-linked Agammaglobulinemia with Delayed Growth.
Mi Jin Kim, Ho Young Kim, Mi Deok Lee, Hong Seung Kim, Young Goo Shin, Choon Hee Chung, Kye Chul Shin
J Korean Endocr Soc. 1999;14(1):153-159.   Published online January 1, 2001
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  • 25 Download
AbstractAbstract PDF
In 1980, the clinical syndrome of X-linked agammaglobulinemia and isolated growth hormone defieiency (XLA/IGHD) was first described by Fleisher. We report here 25-year old man with isolated growth hormone deficiency and X-linked agammaglobulinemia. His height was below the third percentile. He had recurrent sinopulmonary infection, lacked circulating B lympocytes and had deficient growth hormone.
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